چکیده مقاله Cancer prognostication by geometric-bayesian neoplasm entropy signal motility
We have managed to procreatively define spaces of (any) n-dimensions whereby coplanarity is not at all specified by the property of orthogonality or mirror orthogonality.Various pre- and post- metastasistumors are thus stratified topo-algebraically by apportioning specific non-constant polynomials both according to patients’ Quality of Life Index-Cancer Scale (CQOLC) analysis and according to the geometrical statistics tailor-designed by us [mostly --but not everywhere-- based on Bayesian statistics]. Key characteristics of genesis and epigenesis of cancer-prone cellular differentiation, however, increase the level of plasticity handed to multidisciplinary cancer integrative treatment. Frameworks of either scale-free or scale-bound topology of interaction between reality of pain suffered on the one hand, and life quality recursive on the other, has such an entropic distribution that topological categories contrary to traditional wisdom surface to show themselves up as doublets of another alterity —which is not exactly that epitomised through cancer staging categories when human psyche approaches the level of being blown to pieces by the means of end-stage multilateral positing. Thence, there is no reason why we might not expect figures of relatively indifferent significance to be there, emerging from the original non-metastatic dysplastic tissues.They are leveled at perimeter EITHER as CW-complexing with high signaling neoplasm entropy OR as nearly scale-patterned where the integrative oncologist has a full-trees geometry thereof.The treating physician (as the integrative cancer care-provider) does not have to resort to pseudoscience or archetypal practice for seeing through how neoplastic signaling disturbance as common oncogenic mutations disturb the normal functionality of the pathways resulting into disregulated mitogenesis, resistance to pro-apoptotic insults, and some increase in motility; which go to the extreme of turning into other alternatives expressed relatively well through carcinogenic mutations. There is relatively little doubt that the same geometrical topo-statistics could go well with elucidating normal tissue regeneration which has very rarely come to the attention of tissue engineers.