Exploring the Significance of p۵۳ Gene in Lung Cancer: Etiological Factors, Clinical Implications, and Therapeutic Approaches

Lung cancer is a type of cancer that originates in the lungs, which are responsible for breathing. The p۵۳ gene plays a critical crucial role in inhibiting cancer progression by regulating cell growth, DNA repair, and apoptosis. In lung cancer, p۵۳ mutations are common and associated with aggressive tumor growth, chemotherapy resistance, and poor survival outcomes. This article reviews explores the etiological factors and therapeutic approaches related to p۵۳ dysregulation in lung cancer.  Early detection is key for improving treatment response success and patient survival. Research on p۵۳ mutations in lung cancer has provided valuable insights into the molecular mechanisms driving tumorigenesis and treatment response. Targeted therapies for the treatment of lung cancer treatment have shown immense potential by targeting the p۵۳ pathway. It is critical to understand the clinical significance of p۵۳ mutations as they play a crucial role in determining the success of treatment and the patient's prognosis. Personalized treatment approaches must be considered, and future research should focus on developing new targeted therapies, expanding knowledge of p۵۳ mutations in other cancer types, and improving diagnostic tools for to identifying p۵۳ mutations in lung cancer patients. By investing in these areas, we can pave the way for more effective and personalized treatment for lung cancer patients.

کلیدواژه ها:

Lung ، Cancer ، p۵۳ ، Mutation